

Thanks a lot, this is what I’ve been wondering. I know that there’s a link and that cannabis can trigger e.g. psychoses especially if there’s a genetic predisposition, but this is extremely tricky data to work with and to draw inferences from.
They do mention the bidirectional nature and I think that’s where it becomes really hard to model. If you would properly try to isolate the cannabis effect you’d need to identify a cohort of individuals similar to those that got diagnosed and that have (self-reportedly) not consumed cannabis as a control group and then compare these two groups to their overarching population and then determine if the mean difference (if there’s any) is statistically significant.
Here you could argue that this is what they’ve already been doing and I may have a flaw in my thinking, but I think there’s just some control variable missing.
Of course if we knew how to describe such population this would be an easy exercise, but since all we know is that the population is teenagers living in the same area, along with some other demographic metadata this is a limitation that I‘m not sure can be overcome so I don’t blame the authors of the study.
I think that the connection is there in some way or another is an interesting finding and probably a good reason to try and stop teenagers from smoking weed. Personally it also helps me against depression but also I‘m not 14 anymore.






Iceland isn’t in the EU?